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Diabetic muscle infarction
Author(s) -
MacIsaac Richard J,
Jerums George,
Scurrah Lisa
Publication year - 2002
Publication title -
medical journal of australia
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.904
H-Index - 131
eISSN - 1326-5377
pISSN - 0025-729X
DOI - 10.5694/j.1326-5377.2002.tb04793.x
Subject(s) - repatriation , george (robot) , medicine , library science , editor in chief , management , political science , art , art history , law , computer science , economics
THE DIAGNOSIS of diabetic muscle infarction was made retrospectively on clinical grounds and after reviewing the subsequent investigations. The differential diagnoses — muscle strain, rupture, haematoma, myositis, infection, deep venous thrombosis, thrombophlebitis, femoral artery aneurysm, fracture or a connective tissue tumour — were excluded by the clinical presentation, together with the ultrasound, computed tomography (CT) scan and biopsy findings. The CT scan did not reveal a discrete mass, but extensive oedema of one muscle group and sparing of an adjacent muscle group. Histologically, there was evidence of skeletal muscle fibre necrosis, with a variable amount of muscle regeneration and fibrosis. These are the typical features of diabetic muscle infarction. Reports of spontaneous muscle infarction appear to be virtually confined to patients with diabetes. Spontaneous muscle infarction is a rare diabetic complication. There have been fewer than 100 patients reported since 1965.1-12 However, it is becoming more frequently recognised; almost half of the cases have been reported since 1999. It has a predilection for the quadriceps (62%), hip adductors (13%), hamstrings (8%) and hip flexor (2%) muscles. Rarely, the calf and anterior tibial muscles are Diabetic muscle infarction