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registered with the British Boxing Board of Control between the years 1929 and 1955. Only British nationals who had been licensed to box for at least three years were included in the study, and the assessment took place from 1967 onwards. All 250 subjects were traced, and it was possible to examine 224. Eleven had neurological disease which was unlikely to be due to boxing, but 37 (roughly one-sixth of the subjects examined) had a characteristic neurological syndrome, consisting in its typical form of a non-progressive dementia with manifestations of abnormal pyramidal, extrapyramidal and cerebellar function combined in various proportions. The more fights a boxer had, and the heavier the division in which he tought, the greater his risk of developing this traumatic encephalopathy of boxing. The incidence of EEG abnormalities in the boxers was similar to that in a control group of subjects, but this finding is scarcely surprising when one considers how frequently the EEG is normal when recorded some time after the receipt of severe head injuries. No pathological material was included in the study, but by analogy with what is known of the !pathology of head injury, Roberts was able to infer a possible neuropathological basis for the traumatic encephalopathy syndrome. As well as the neurological disorders mentioned above, Roberts found an appreciable incidence of visual and neuro-otclogical disturbances in his subjects, but did not go into these aspects at length.