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Reduced gray matter volume in the posterior cingulate cortex (PCC) has recently been found in patients with non-rapid eye movement (NREM) parasomnia, providing a neuroanatomical substrate for the arousal state dissociation. It remains unclear whether PCC changes in NREM parasomnias might also play a role in cognitive or affective dysfunction in these patients. The aim of this exploratory study was to investigate neurobehavioral correlates of PCC abnormalities in patients with NREM parasomnia.

Increased behavioral inhibition trait and negative stress coping in non–rapid eye movement parasomnias