Open AccessCONSEQUENCES OF INTERMITTENT FASTING FOR NEUROPLASTICITY:
Author(s) -
Idrys Henrique Leite Guedes,
Anna Luisa de Souza Holanda,
Daniela Vieira da Silveira Santos,
Grazielle Romaneto Marques,
João Carlos de Oliveira Dutra,
Leonardo Franco de Almeida
Publication year - 2021
Publication title -
health and society
Language(s) - English
Resource type - Journals
ISSN - 2763-5724
DOI - 10.51249/hes01.03.2021.350
Subject(s) - neuroplasticity , intermittent fasting , neuroscience , biology , energy metabolism , endocrinology
Neuroplasticity occurs from human development until the end of senility and, despite the dependence on food, the regulation of the diet on neurons and nerve synapses is not restricted to the availability of nutrients. In turn, intermittent fasting, as a dietary strategy, represents not only a modern technique for weight loss, but also an important historical factor for the occurrence of persistent organic adaptations. Based on this interrelation, evidence was collected from databases that associated, positively or negatively, neuroplasticity with intermittent fasting in humans. From the review of the selected articles, the various ways in which intermittent fasting affects neuroplasticity, influencing energy metabolism, triggering signaling pathways, modulating components of the immune system and altering cellular components were identified. All of these changes create a favorable environment for the emergence and maintenance of neurons, as well as for the formation of new nerve synapses.