Increased apoptosis, tumor necrosis factor-α, and DNA damage attenuated by 3',4'-dihydroxyflavonol in rats with brain İschemia-reperfusion | Zendy

Dervis Dasdelen | Zendy; Merve Solmaz | Zendy; Esma Menevşe | Zendy; Rasim Moğulkoç | Zendy; Abdulkerim Kasim Baltaci | Zendy; Ender Erdoğan | Zendy

Open Access

Increased apoptosis, tumor necrosis factor-α, and DNA damage attenuated by 3',4'-dihydroxyflavonol in rats with brain İschemia-reperfusion

Author(s) -

Dervis Dasdelen,

Merve Solmaz,

Esma Menevşe,

Rasim Moğulkoç,

Abdulkerim Kasim Baltaci,

Ender Erdoğan

Publication year - 2021

Publication title -

indian journal of pharmacology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.286

H-Index - 59

eISSN - 1998-3751

pISSN - 0253-7613

DOI - 10.4103/ijp.ijp_727_20

Subject(s) - apoptosis , dna damage , tumor necrosis factor alpha , tumor necrosis factor α , necrosis , dna , brain damage , chemistry , microbiology and biotechnology , biology , pharmacology , cancer research , medicine , pathology , immunology , neuroscience , genetics

This research was aimed to find out the effects of 3',4'-dihydroxyflavonol (DiOHF) on apoptosis, DNA damage, and tumor necrosis factor-α (TNF-α) levels in the frontal cortex of rats with induced experimental brain ischemi reperfusion.

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