Cigarette smoke condensate could promote human bronchial epithelial BEAS-2B cell migration through shifting neprilysin trafficking | Zendy

Kun Yang | Zendy; Chuanfeng Zhang | Zendy; Lei Sun | Zendy; Dong Li | Zendy; Xin Hong | Zendy

Open Access

Cigarette smoke condensate could promote human bronchial epithelial BEAS-2B cell migration through shifting neprilysin trafficking

Author(s) -

Kun Yang,

Chuanfeng Zhang,

Lei Sun,

Dong Li,

Xin Hong

Publication year - 2018

Publication title -

journal of cancer research and therapeutics

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.475

H-Index - 39

eISSN - 0973-1482

pISSN - 1998-4138

DOI - 10.4103/0973-1482.183182

Subject(s) - neprilysin , chemistry , microbiology and biotechnology , internalization , cell , cell growth , cell migration , lysosome , cancer research , biology , biochemistry , enzyme

Recent studies have suggested neprilysin (NEP) play a key role in cigarette smoke-induced nonsmall-cell lung carcinoma; however, the detailed mechanism was still unclear. Here, we employed in vitro human bronchial epithelial BEAS-2B cells to investigate whether and how NEP involved in cigarette smoke condensate (CSC)-induced cancer occurrence.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research