Open AccessIsolated symmetrical bilateral basal ganglia T2 hyperintensity in carbon monoxide poisoning
Author(s) -
S. Subhaschandra,
W. Jatishwor,
Th. Suraj
Publication year - 2008
Publication title -
annals of indian academy of neurology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.427
H-Index - 31
eISSN - 1998-3549
pISSN - 0972-2327
DOI - 10.4103/0972-2327.44563
Subject(s) - carbon monoxide poisoning , hyperintensity , medicine , basal ganglia , co poisoning , basal (medicine) , carbon monoxide , medical history , clinical history , clinical diagnosis , pathology , magnetic resonance imaging , radiology , poison control , intensive care medicine , surgery , medical emergency , central nervous system , biochemistry , chemistry , insulin , catalysis
Carbon monoxide poisoning is not uncommon during the winter months. To make a diagnosis, strong clinical suspicion and acumen, and history of the exposure are necessary. Many a time, the presenting complaints may fail to help reach a diagnosis, in the absence of history. Imaging plays a role in the diagnosis of brain injury with the characteristic features, which are correlated with the clinical profile. Isolated bilateral basal ganglia injury revealing T2 hyperintensity in MRI may be observed in acute carbon monoxide poisoning.