Open AccessSudden decrease in end-tidal carbon-dioxide in a neonate undergoing surgery for type B interrupted aortic arch
Author(s) -
Satyajeet Misra,
Thomas Koshy,
Divya Amol Chandran Mahaldar
Publication year - 2011
Publication title -
annals of cardiac anaesthesia/annals of cardiac anaesthesia
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.42
H-Index - 27
eISSN - 0974-5181
pISSN - 0971-9784
DOI - 10.4103/0971-9784.84020
Subject(s) - interrupted aortic arch , medicine , cardiology , perfusion , anesthesia , aortic arch , ventilation (architecture) , aorta , mechanical engineering , engineering
Interruption of the aortic arch is a rare anomaly affecting 1% of children with congenital heart disease. The systemic circulation is ductal dependent and is determined principally by the ratio of the resistances in the systemic and the pulmonary vascular bed. Any increase in the pulmonary vascular resistance may increase the dead space ventilation due to acute pulmonary hypoperfusion. We report a case where sudden decreases in the end-tidal carbon-dioxide due to pulmonary hypoperfusion mimicked accidental endotracheal tube extubation in an infant undergoing repair of interrupted aortic arch.