Open AccessThe contribution of postnatal steroid administration to early brain damage in preterm babies with bronchopulmonary dysplasia
Author(s) -
Sabahattin Ertuğrul,
Savaş Mert Darakçı,
İbrahim Kaplan,
İlyas Yolbaş,
İbrahim Değer,
Sibel Tanrıverdi Yilmaz,
Şerafettin Aktaş
Publication year - 2021
Publication title -
turkish journal of medical sciences
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.277
H-Index - 27
eISSN - 1303-6165
pISSN - 1300-0144
DOI - 10.3906/sag-2101-295
Subject(s) - bronchopulmonary dysplasia , medicine , enolase , glial fibrillary acidic protein , dexamethasone , gestation , cohort , brain damage , pediatrics , gastroenterology , gestational age , pregnancy , immunohistochemistry , genetics , biology
Postnatal corticosteroids are commonly used to treat bronchopulmonary dysplasia (BPD). We aimed to show whether S100 calcium-binding B (S100B), neuron-specific enolase (NSE), Tau protein or microtubule-associated protein tau (MAPT), and glial fibrillary acid protein (GFAP) levels would provide any evidence of early neurological damage in premature infants receiving postnatal low dose dexamethasone therapy for BPD treatment.