
NaV1.5 sodium channels increase breast cancer cell invadopodial activity by both controlling Src kinase-dependent F-actin polymerization and promoting NHE-1-dependent proton efflux and extracellular matrix digestion
Author(s) -
Lucie Brisson,
Virginie Driffort,
Lauriane Benoist,
Mallorie Poët,
Laurent Counillon,
Ester Antelmi,
Rosa Rubino,
Pierre Besson,
Fabien Labbal,
Stéphan Chevalier,
Stephan J. Reshkin,
J. Goré,
Sébastien Roger
Publication year - 2014
Publication title -
frontiers in pharmacology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.384
H-Index - 86
ISSN - 1663-9812
DOI - 10.3389/conf.fphar.2014.61.00011
Subject(s) - invadopodia , cancer cell , extracellular matrix , microbiology and biotechnology , actin , extracellular , chemistry , cancer research , biology , cancer , genetics