Open AccessAPOLD1 loss causes endothelial dysfunction involving cell junctions, cytoskeletal architecture, and Weibel-Palade bodies, while disrupting hemostasis
Author(s) -
Simon Stritt,
Paquita Nurden,
Alan T. Nurden,
JeanFrançois Schved,
JeanClaude Bordet,
M Roux,
MarieChristine Alessi,
DavidAlexandre Trégouët,
Taija Mäkinen,
Muriel GiansilyBlaizot
Publication year - 2022
Publication title -
haematologica
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.782
H-Index - 142
eISSN - 1592-8721
pISSN - 0390-6078
DOI - 10.3324/haematol.2022.280816
Subject(s) - microbiology and biotechnology , biology , endothelial stem cell , von willebrand factor , endothelial dysfunction , endothelium , vascular endothelial growth factor b , immunology , platelet , cancer research , vascular endothelial growth factor a , genetics , endocrinology , vascular endothelial growth factor , in vitro , vegf receptors