Open AccessNeurogenic Pulmonary Edema
Author(s) -
Carlos Eduardo Romeu de Almeida,
Eberval Gadelha Figueiredo,
Bernardo Assumpção de Mônaco,
Arthur Maynart Pereira Oliveira,
Manoel Jacobsen Teixeira
Publication year - 2018
Publication title -
jornal brasileiro de neurocirurgia
Language(s) - English
Resource type - Journals
eISSN - 2446-6786
pISSN - 0103-5118
DOI - 10.22290/jbnc.v21i1.817
Subject(s) - medicine , anesthesia , pulmonary edema , subarachnoid hemorrhage , subarachnoid space , edema , vasoconstriction , lung , surgery , cerebrospinal fluid
Neurogenic Pulmonary Edema (NPE) is defined as the acute onset of dyspnea or a decrease in PaO2/FiO2 ratio, following an acute central nervous system (CNS) insult, in the absence of other obvious causes of lung injury. The most important cause of NPE is subarachnoid hemorrhage (SAH), followed by cerebraltrauma and epilepsy. The incidence of NPE after SAH in the literature may vary from 4 to 23% in greater studies, with SAH accounting for 43-73% of cases of NPE. It is postulated that an excessive adrenergic discharge would lead to pulmonary vasoconstriction and a rapid increase in pulmonary capillaryhydrostatic pressure, thus promoting fluid leakage to the alveolar space. NPE is generally treated in a supportive and conservative fashion, and patient management should be focused in the primary insult.