Dexmedetomidine Protects Human Renal Tubular Epithelial HK-2 Cells against Hypoxia/Reoxygenation Injury by Inactivating Endoplasmic Reticulum Stress Pathway. | Zendy

Mingyu Zhai | Zendy; Mingming Han | Zendy; Xiang Huang | Zendy; Fang Kang | Zendy; Cheng Yang | Zendy; Juan Li | Zendy

Open Access

Dexmedetomidine Protects Human Renal Tubular Epithelial HK-2 Cells against Hypoxia/Reoxygenation Injury by Inactivating Endoplasmic Reticulum Stress Pathway.

Author(s) -

Mingyu Zhai,

Mingming Han,

Xiang Huang,

Fang Kang,

Cheng Yang,

Juan Li

Publication year - 2021

Publication title -

doaj (doaj: directory of open access journals)

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.712

H-Index - 29

eISSN - 2228-5814

pISSN - 2228-5806

DOI - 10.22074/cellj.2021.7220

Subject(s) - apoptosis , chop , viability assay , dexmedetomidine , biology , hypoxia (environmental) , endoplasmic reticulum , western blot , microbiology and biotechnology , flow cytometry , endocrinology , mtt assay , andrology , medicine , pharmacology , biochemistry , chemistry , gene , organic chemistry , sedation , oxygen

The study was aimed to investigate the effects and potential mechanisms of Dexmedetomidine (Dex) on hypoxia/reoxygenation (H/R) injury in human renal tubular epithelial HK-2 cells.

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