Mechanism of oxidative stress p38MAPK-SGK1 signaling axis in experimental autoimmune encephalomyelitis (EAE) | Zendy

Liang Wang | Zendy; Bin Li | Zendy; Moyuan Quan | Zendy; Li Lin | Zendy; Yuan Chen | Zendy; Guo-Jun Tan | Zendy; Jing Zhang | Zendy; Xiaopeng Liu | Zendy; Li Guo | Zendy

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Mechanism of oxidative stress p38MAPK-SGK1 signaling axis in experimental autoimmune encephalomyelitis (EAE)

Author(s) -

Liang Wang,

Bin Li,

Moyuan Quan,

Li Lin,

Yuan Chen,

Guo-Jun Tan,

Jing Zhang,

Xiaopeng Liu,

Li Guo

Publication year - 2017

Publication title -

oncotarget

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.373

H-Index - 127

ISSN - 1949-2553

DOI - 10.18632/oncotarget.17057

Subject(s) - experimental autoimmune encephalomyelitis , oxidative stress , multiple sclerosis , pathogenesis , immunology , inflammation , immune system , sgk1 , signal transduction , medicine , biology , glucocorticoid , microbiology and biotechnology , endocrinology

Multiple sclerosis (MS), a complex disease associated with multifocal demyelination of the central nervous system and poorly understood etiology. It has been previously indicated that many factors, including oxidative stress and p38MAPK-SGK1 pathway, contribute to the pathogenesis of MS.

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