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Endothelial nitric oxide synthase deficiency influences normal cell cycle progression and apoptosis in trabecular meshwork cells
Author(s) -
Qiong Liao,
Yanming Huang,
Wenting Fan,
Chan Li,
Yun–Chul Hong
Publication year - 2016
Publication title -
international journal of ophthalmology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.634
H-Index - 29
eISSN - 2227-4898
pISSN - 2222-3959
DOI - 10.18240/ijo.2016.06.01
Subject(s) - medicine , trabecular meshwork , nitric oxide synthase , apoptosis , endothelial nitric oxide synthase , nitric oxide , microbiology and biotechnology , ophthalmology , glaucoma , biochemistry , enos , biology
To clarify how the endothelial nitric oxide synthase (eNOS, NOS3) make effect on outflow facility through the trabecular meshwork (TM).

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