Glutathione reductase system and acetylcholinesterase activity of erythrocytes in ischemic heart disease | Zendy

V. N. Fatenkov | Zendy; N. A. Klenova | Zendy; E. I. Seleznev | Zendy; A. S. Stegunin | Zendy; R. A. Timerbulatov | Zendy

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Glutathione reductase system and acetylcholinesterase activity of erythrocytes in ischemic heart disease

Author(s) -

V. N. Fatenkov,

N. A. Klenova,

E. I. Seleznev,

A. S. Stegunin,

R. A. Timerbulatov

Publication year - 1984

Publication title -

kazan medical journal

Language(s) - English

Resource type - Journals

eISSN - 2587-9359

pISSN - 0368-4814

DOI - 10.17816/kazmj88727

Subject(s) - glutathione reductase , glutathione , acetylcholinesterase , hypoxia (environmental) , enzyme , homeostasis , biochemistry , chemistry , reductase , lactate dehydrogenase , dehydrogenase , pharmacology , medicine , endocrinology , biology , glutathione peroxidase , oxygen , organic chemistry

In the mechanism of hypoxia developing in ischemic heart disease (CHD), along with other factors, structural and functional disorders of the organization of erythrocytes are important [1, 8, 12]. The glutathione reductase system, including glucose-6-phosphate dehydrogenase (CF 1.1.1.49), glutathione reductase (CF 1.6.4.2) and reduced glutathione, plays a certain role in maintaining erythrocyte homeostasis (ER) [15]. The experiment shows the relationship of these systems with the activity of membrane-bound enzymes and, in particular, with acetylcholinesterase (CF. 3.1.1.7.) [4].

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