Rosiglitazone Inhibits Acyl-CoA Synthetase Activity and Fatty Acid Partitioning to Diacylglycerol and Triacylglycerol via a Peroxisome Proliferator-Activated Receptor- -Independent Mechanism in Human Arterial Smooth Muscle Cells and Macrophages | Zendy

Benyamin Askari | Zendy; J. E. Kanter | Zendy; A. M. Sherrid | Zendy; D. L. Golej | Zendy; A. T. Bender | Zendy; J. Liu | Zendy; W. A. Hsueh | Zendy; J. A. Beavo | Zendy; R. A. Coleman | Zendy; Karin Bornfeldt | Zendy

Open Access

Rosiglitazone Inhibits Acyl-CoA Synthetase Activity and Fatty Acid Partitioning to Diacylglycerol and Triacylglycerol via a Peroxisome Proliferator-Activated Receptor- -Independent Mechanism in Human Arterial Smooth Muscle Cells and Macrophages

Author(s) -

Benyamin Askari,

J. E. Kanter,

A. M. Sherrid,

D. L. Golej,

A. T. Bender,

J. Liu,

W. A. Hsueh,

J. A. Beavo,

R. A. Coleman,

Karin Bornfeldt

Publication year - 2007

Publication title -

carolina digital repository (university of north carolina at chapel hill)

Language(s) - English

DOI - 10.17615/xbnv-2n71

Subject(s) - peroxisome , diacylglycerol kinase , chemistry , rosiglitazone , peroxisome proliferator activated receptor , acyl coa , mechanism (biology) , biochemistry , receptor , peroxisome proliferator activated receptor alpha , fatty acid , microbiology and biotechnology , medicine , endocrinology , biology , enzyme , nuclear receptor , transcription factor , protein kinase c , gene , philosophy , epistemology

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