Open AccessApoptosis promotes a caspase-induced amino-terminal truncation of IκBα that functions as a stable inhibitor of NF-kB
Author(s) -
J.Y. Reuther,
A.S. Baldwin
Publication year - 1999
Publication title -
carolina digital repository (university of north carolina at chapel hill)
Language(s) - English
DOI - 10.17615/nhpm-fj58
Subject(s) - apoptosis , truncation (statistics) , terminal (telecommunication) , chemistry , microbiology and biotechnology , caspase , caspase 3 , inhibitor of apoptosis , caspase 8 , biology , programmed cell death , biochemistry , mathematics , computer science , telecommunications , statistics