Open AccessClosure of VDAC causes oxidative stress and accelerates the Ca2+-induced mitochondrial permeability transition in rat liver mitochondria
Author(s) -
Andrey P. Tikunov,
C. Bryce Johnson,
Peter Pediaditakis,
Nikolai Markevich,
Jeffrey M. Macdonald,
John J. Lemasters,
Ekhson Holmuhamedov
Publication year - 2010
Publication title -
carolina digital repository (university of north carolina at chapel hill)
Language(s) - English
DOI - 10.17615/gfb2-1w14
Subject(s) - voltage dependent anion channel , mitochondrial permeability transition pore , mitochondrion , oxidative stress , oxidative phosphorylation , microbiology and biotechnology , chemistry , closure (psychology) , permeability (electromagnetism) , biophysics , biology , biochemistry , apoptosis , membrane , programmed cell death , bacterial outer membrane , political science , escherichia coli , gene , law
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