Shiga toxin 2-induced intestinal pathology in infant rabbits is A-subunit dependent and responsive to the tyrosine kinase and potential ZAK inhibitor imatinib
Author(s) -
Samuel M. Stone,
Cheleste M. Thorpe,
Amrita Ahluwalia,
Arlin B. Rogers,
Fumiko Obata,
Aimee E. Vozenilek,
Glynis L. Kolling,
Anne Kane,
Bruce E. Magun,
Dakshina M. Jandhyala
Publication year - 2012
Publication title -
carolina digital repository (university of north carolina at chapel hill)
Language(s) - English
DOI - 10.17615/fd7k-1c32
Subject(s) - imatinib , toxin , protein subunit , tyrosine kinase inhibitor , tyrosine kinase , shiga toxin , tyrosine , biology , medicine , microbiology and biotechnology , pathology , immunology , receptor , biochemistry , escherichia coli , myeloid leukemia , cancer , gene
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