Premium
Toxicity of Hydrogen Sulfide to Various Life History Stages of Bluegill (Lepomis macrochirus)
Author(s) -
Smith Lloyd L.,
Oseid Donavon M.,
Kimball Gary L.,
ElKandelgy Sayed M.
Publication year - 1976
Publication title -
transactions of the american fisheries society
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.696
H-Index - 86
eISSN - 1548-8659
pISSN - 0002-8487
DOI - 10.1577/1548-8659(1976)105<442:tohstv>2.0.co;2
Subject(s) - liter , lepomis macrochirus , toxicity , chronic toxicity , zoology , acute toxicity , toxicology , acclimatization , chemistry , biology , endocrinology , ecology , fishery , fish <actinopterygii> , organic chemistry
Bluegill (Lepomis macrochirus Rafinesque) eggs, fry, juveniles, and adults were exposed to H 2 S concentrations to determine acute toxicity. Seventy‐two‐hour LC50 for eggs was 0.0190 mg/liter; 96‐h LC50 for 35‐day‐old fry was 0.0131 mg/liter, for juveniles 0.0478 mg/liter, and for adults 0.0448 mg/liter. Exposure to lower levels of H 2 S resulted in some acclimation. Chronic exposure to sublethal levels of H 2 S for up to 826 days resulted in no egg deposition at 0.0022 mg/liter and reduced deposition after 97 days at 0.0010 mg/liter. Growth was adversely affected at levels from 0.0031 to 0.0107 mg/liter H 2 S depending on the life history stage at which chronic exposure was started. When exposure was started with eggs, the lower level retarded growth. Food consumption was reduced at 0.0085 mg/liter H 2 S. Time to anesthesia with MS:222 was reduced at levels from 0.0014 to 0.0031 mg/liter H 2 S. The most sensitive stage to acute toxicity was the feeding fry and to chronic toxicity was the spawning adult.