METTL3 promotes inflammation and cell apoptosis in a pediatric pneumonia model by regulating EZH2 | Zendy

Xiaoqing Yang | Zendy; Ying Yang | Zendy; Yi Wu | Zendy; Meijiao Fu | Zendy

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METTL3 promotes inflammation and cell apoptosis in a pediatric pneumonia model by regulating EZH2

Author(s) -

Xiaoqing Yang,

Ying Yang,

Yi Wu,

Meijiao Fu

Publication year - 2021

Publication title -

allergologia et immunopathologia

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.483

H-Index - 37

eISSN - 1578-1267

pISSN - 0301-0546

DOI - 10.15586/aei.v49i5.445

Subject(s) - gene knockdown , western blot , apoptosis , viability assay , medicine , flow cytometry , cell , cancer research , cell growth , immunology , biology , biochemistry , gene

In recent times, the medical science has developed by leaps and bounds, however, the molecular mechanism of pediatric pneumonia is still unclear. Although prior researches have shown that methyltransferase-like 3 (METTL3) is up-regulated in a variety of inflammatory diseases, its role and mechanism has been rarely studied in pediatric pneumonia, and need to be defined elaborately.

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