Open AccessCorrection: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms
Author(s) -
Sandra Meidute Abaraviciene,
Ingmar Lundquist,
Juris Galvanovskis,
Erik Flodgren,
Björn Olde,
Albert Salehi
Publication year - 2008
Publication title -
plos one
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.99
H-Index - 332
ISSN - 1932-6203
DOI - 10.1371/annotation/21d1a1f6-35e9-4935-a8d0-387c057f1469
Subject(s) - signal transduction , thiazolidinedione , free fatty acid receptor 1 , transduction (biophysics) , medicine , microbiology and biotechnology , chemistry , endocrinology , pharmacology , biology , receptor , biochemistry , diabetes mellitus , type 2 diabetes , agonist