Open AccessCorrelation between circulating endothelial cell level and acute respiratory distress syndrome in postoperative patients
Author(s) -
Min Peng,
Yan Qing-he,
Ying Gao,
Zhen Zhang,
Ying Zhang,
Yifeng Wang,
He-Ning Wu
Publication year - 2021
Publication title -
world journal of clinical cases
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.368
H-Index - 10
ISSN - 2307-8960
DOI - 10.12998/wjcc.v9.i32.9731
Subject(s) - ards , medicine , diffuse alveolar damage , lung , pulmonary edema , edema , respiratory distress , acute respiratory distress , pathology , circulatory system , respiratory system , endothelial stem cell , cardiology , anesthesia , in vitro , biochemistry , chemistry
Acute respiratory distress syndrome (ARDS) is injury of alveolar epithelial cells and capillary endothelial cells caused by various factors, including endogenous and exogenous lung factors, leading to diffuse pulmonary interstitial and alveolar edema, and acute respiratory failure. ARDS involves alveolar epithelial cells and pulmonary interstitial capillary endothelial cells. Circulating endothelial cells (CECs) are the only marker that directly reflects vascular endothelial injury in vivo. There have been few studies on the correlation between peripheral blood CECs and ARDS at home and abroad. The lungs are the organs with the highest capillary density and the most endothelial cells, thus, it is speculated that when ARDS occurs, CECs are stimulated and damaged, and released into the circulatory system.