Do the Actions of Glucagon-Like Peptide-1 on Gastric Emptying, Appetite, and Food Intake Involve Release of Amylin in Humans? | Zendy

Meena Asmar | Zendy; M. Bache | Zendy; Filip K. Knop | Zendy; Sten Madsbad | Zendy; Jens J. Holst | Zendy

Open Access

Do the Actions of Glucagon-Like Peptide-1 on Gastric Emptying, Appetite, and Food Intake Involve Release of Amylin in Humans?

Author(s) -

Meena Asmar,

M. Bache,

Filip K. Knop,

Sten Madsbad,

Jens J. Holst

Publication year - 2010

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2009-2133

Subject(s) - amylin , gastric emptying , appetite , medicine , endocrinology , postprandial , glucagon , incretin , glucagon like peptide 1 , peptide yy , crossover study , insulin , meal , diabetes mellitus , type 2 diabetes , stomach , neuropeptide , islet , placebo , receptor , neuropeptide y receptor , alternative medicine , pathology

Amylin, cosecreted with insulin, has like glucagon-like peptide-1 (GLP-1) been reported to inhibit glucagon secretion, delay gastric emptying, and reduce appetite and food intake. We investigated whether the effects of GLP-1 on gastric emptying, appetite, and food intake are mediated directly or indirectly via release of amylin.

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