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Recruited monocytes modulate malaria‐induced lung injury through CD36‐mediated clearance of sequestered infected erythrocytes
Author(s) -
Lagassé H. A. Daniel,
Anidi Ifeanyi U.,
Craig John M.,
Limjunyawong Nathachit,
Poupore Amy K.,
Mitzner Wayne,
Scott Alan L.
Publication year - 2016
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.4hi0315-130rrr
Subject(s) - plasmodium berghei , lung , biology , cd36 , immunology , monocyte , immune system , malaria , ccr2 , inflammation , bone marrow , receptor , medicine , chemokine , chemokine receptor , biochemistry
Recruited monocytes limit ALI by clearing parasite‐infected cells via a CD36‐mediated mechanism.

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