Estrogen anti‐inflammatory activity on human monocytes is mediated through cross‐talk between estrogen receptor ERα36 and GPR30/GPER1 | Zendy

Pelekanou Vasiliki | Zendy; Kampa Marilena | Zendy; Kiagiadaki Foteini | Zendy; Deli Alexandra | Zendy; Theodoropoulos Panayiotis | Zendy; Agrogiannis George | Zendy; Patsouris Efstratios | Zendy; Tsapis Andreas | Zendy; Castanas Elias | Zendy; Notas George | Zendy

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Estrogen anti‐inflammatory activity on human monocytes is mediated through cross‐talk between estrogen receptor ERα36 and GPR30/GPER1

Author(s) -

Pelekanou Vasiliki,

Kampa Marilena,

Kiagiadaki Foteini,

Deli Alexandra,

Theodoropoulos Panayiotis,

Agrogiannis George,

Patsouris Efstratios,

Tsapis Andreas,

Castanas Elias,

Notas George

Publication year - 2016

Publication title -

journal of leukocyte biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.819

H-Index - 191

eISSN - 1938-3673

pISSN - 0741-5400

DOI - 10.1189/jlb.3a0914-430rr

Subject(s) - estrogen receptor , gper , estrogen receptor beta , estrogen , biology , estrogen receptor alpha , receptor , estrogen related receptor gamma , monocyte , cancer research , microbiology and biotechnology , endocrinology , medicine , biochemistry , immunology , cancer , breast cancer , genetics

Ligand‐activated ER, ERα36, inhibits LPS‐induced IL‐6 expression in human monocytes via inhibition of NFκB transcriptional activity, in a GPR30/GPER1‐dependent manner.

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