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Galectin‐3–null mice display defective neutrophil clearance during acute inflammation
Author(s) -
Wright Rachael D,
Souza Patricia R.,
Flak Magdalena B.,
Thedchanamoorthy Prasheetha,
Norling Lucy V.,
Cooper Dianne
Publication year - 2017
Publication title -
journal of leukocyte biology
Language(s) - Uncategorized
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.3a0116-026rr
Subject(s) - efferocytosis , inflammation , biology , phagocytosis , immunology , peritoneum , proinflammatory cytokine , galectin , s100a9 , peritonitis , galectin 3 , macrophage , in vitro , biochemistry , genetics , anatomy
Expression of galectin‐3 by exudated neutrophils drives neutrophil apoptosis and clearance in a model of self‐resolving peritonitis.