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An increased expression of PI‐PLCβ1 is associated with myeloid differentiation and a longer response to azacitidine in myelodysplastic syndromes
Author(s) -
Cocco Lucio,
Finelli Carlo,
Mongiorgi Sara,
Clissa Cristina,
Russo Domenico,
Bosi Costanza,
Quaranta Marilisa,
Malagola Michele,
Parisi Sarah,
Stanzani Marta,
Ramazzotti Giulia,
Mariani Giulia A.,
Billi Anna Maria,
Manzoli Lucia,
Follo Matilde Y.
Publication year - 2015
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.2ma1114-541r
Subject(s) - azacitidine , myelodysplastic syndromes , myeloid , biology , hypomethylating agent , medicine , myeloid leukemia , cancer research , oncology , immunology , gene expression , dna methylation , gene , bone marrow , genetics
Nuclear PI‐PLCβ1 expression is differentially modulated in low‐ and high‐risk MDS patients responding to azacitidine, contributing to the activation of myeloid differentiation.