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Neutrophils rapidly transit inflamed lymphatic vessel endothelium via integrin‐dependent proteolysis and lipoxin‐induced junctional retraction
Author(s) -
Rigby David A.,
Ferguson David J. P.,
Johnson Louise A.,
Jackson David G.
Publication year - 2015
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.1hi0415-149r
Subject(s) - biology , microbiology and biotechnology , integrin , endothelium , immunology , lymphangiogenesis , lymphatic system , lymphatic endothelium , high endothelial venules , inflammation , endothelial stem cell , lymph , pathology , in vitro , receptor , medicine , biochemistry , genetics , cancer , metastasis , endocrinology
Adhesion of neutrophils to inflamed lymphatic endothelium via β2 integrin triggers protease and lipoxin activities promoting junctional retraction and rapid transmigration.