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The granulocyte‐colony stimulating factor receptor (G‐CSFR) interacts with retinoic acid receptors (RARs) in the regulation of myeloid differentiation
Author(s) -
Chee Lynette C. Y.,
Hendy Jean,
Purton Louise E.,
McArthur Grant A.
Publication year - 2013
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.1211609
Subject(s) - biology , granulocyte colony stimulating factor receptor , retinoic acid , receptor , myeloid , granulocyte , granulocyte macrophage colony stimulating factor receptor , retinoic acid receptor , microbiology and biotechnology , colony stimulating factor , granulocyte colony stimulating factor , myeloid cells , immunology , cancer research , biochemistry , genetics , gene , haematopoiesis , macrophage colony stimulating factor , in vitro , stem cell , chemotherapy , macrophage
Residual granulopoiesis persists in mice lacking both G‐CSFR and RARα, or RARγ; G‐CSFR is required for granulopoietic expansion seen with loss of RARγ.