The granulocyte‐colony stimulating factor receptor (G‐CSFR) interacts with retinoic acid receptors (RARs) in the regulation of myeloid differentiation | Zendy

Chee Lynette C. Y. | Zendy; Hendy Jean | Zendy; Purton Louise E. | Zendy; McArthur Grant A. | Zendy

Premium

The granulocyte‐colony stimulating factor receptor (G‐CSFR) interacts with retinoic acid receptors (RARs) in the regulation of myeloid differentiation

Author(s) -

Chee Lynette C. Y.,

Hendy Jean,

Purton Louise E.,

McArthur Grant A.

Publication year - 2013

Publication title -

journal of leukocyte biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.819

H-Index - 191

eISSN - 1938-3673

pISSN - 0741-5400

DOI - 10.1189/jlb.1211609

Subject(s) - biology , granulocyte colony stimulating factor receptor , retinoic acid , receptor , myeloid , granulocyte , granulocyte macrophage colony stimulating factor receptor , retinoic acid receptor , microbiology and biotechnology , colony stimulating factor , granulocyte colony stimulating factor , myeloid cells , immunology , cancer research , biochemistry , genetics , gene , haematopoiesis , macrophage colony stimulating factor , in vitro , stem cell , chemotherapy , macrophage

Residual granulopoiesis persists in mice lacking both G‐CSFR and RARα, or RARγ; G‐CSFR is required for granulopoietic expansion seen with loss of RARγ.

This content is not available in your region!

Continue researching here.

Having issues? You can contact us here

Accelerating Research