Premium
CCL22 regulates experimental autoimmune encephalomyelitis by controlling inflammatory macrophage accumulation and effector function
Author(s) -
Dogan RukiyeNazan E.,
Long Nancy,
Forde Eileen,
Dennis Kristen,
Kohm Adam P.,
Miller Stephen D.,
Karpus William J.
Publication year - 2011
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0810442
Subject(s) - experimental autoimmune encephalomyelitis , immunology , biology , ccl22 , adoptive cell transfer , t cell , effector , macrophage , chemokine , cytokine , encephalomyelitis , immune system , multiple sclerosis , in vitro , cxcl10 , biochemistry
CCL22 functions to regulate EAE development through CNS macrophage chemoattraction and the balance of macrophage inflammatory and anti‐inflammatory cytokine expression.