Premium
Mycobacterium tuberculosis induces IL‐17A responses through TLR4 and dectin‐1 and is critically dependent on endogenous IL‐1
Author(s) -
Veerdonk Frank L.,
Teirlinck Anne C.,
Kleinnijenhuis Johanneke,
Kullberg Bart Jan,
Crevel Reinout,
Meer Jos W. M.,
Joosten Leo A. B.,
Netea Mihai G.
Publication year - 2010
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0809550
Subject(s) - tlr4 , biology , tlr2 , proteases , endogeny , mycobacterium tuberculosis , signal transduction , mapk/erk pathway , interleukin 17 , microbiology and biotechnology , receptor , intracellular , immunology , cytokine , tuberculosis , enzyme , biochemistry , medicine , pathology
IL‐17A production induced by Mycobacterium tuberculosis through TLR4 and dectin‐1 is dependent on the steps necessary for the induction of endogenous IL‐1β.
Accelerating Research
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom
Address
John Eccles HouseRobert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom