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Opioid‐induced chemokine expression requires NF‐κB activity: the role of PKCζ
Author(s) -
Happel Christine,
Kutzler Michele,
Rogers Thomas J.
Publication year - 2011
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0710402
Subject(s) - damgo , proinflammatory cytokine , chemokine , microbiology and biotechnology , biology , ccl5 , nf κb , protein kinase c , signal transduction , agonist , opioid receptor , receptor , immunology , immune system , inflammation , t cell , biochemistry , il 2 receptor
Mu opioid receptor activation induces the expression of chemokine CCL2 through a pro‐inflammatory, PKCζ‐dependent, NF‐κB pathway.
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