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Phosphoinositide 3‐kinase γ plays a critical role in bleomycin‐induced pulmonary inflammation and fibrosis in mice
Author(s) -
Russo Remo C.,
Garcia Cristiana C.,
Barcelos Lucíola S.,
Rachid Milene A.,
Guabiraba Rodrigo,
Roffê Ester,
Souza Adriano L. S.,
Sousa Lirlândia P.,
Mirolo Massimiliano,
Doni Andrea,
Cassali Geovanni D.,
Pinho Vanessa,
Locati Massimo,
Teixeira Mauro M.
Publication year - 2011
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0610346
Subject(s) - bleomycin , angiogenesis , fibrosis , pulmonary fibrosis , inflammation , pi3k/akt/mtor pathway , cancer research , biology , immunology , pathology , medicine , microbiology and biotechnology , signal transduction , chemotherapy , genetics
PI3Kγ is important to drive the fibrotic processes by regulation of pulmonary inflammation and angiogenesis in mice, suggesting PI3Kγ as therapeutic target for pulmonary fibrosis.