GCN5 is essential for IRF‐4 gene expression followed by transcriptional activation of Blimp‐1 in immature B cells | Zendy

Kikuchi Hidehiko | Zendy; Nakayama Masami | Zendy; Kuribayashi Futoshi | Zendy; ImajohOhmi Shinobu | Zendy; Nishitoh Hideki | Zendy; Takami Yasunari | Zendy; Nakayama Tatsuo | Zendy

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GCN5 is essential for IRF‐4 gene expression followed by transcriptional activation of Blimp‐1 in immature B cells

Author(s) -

Kikuchi Hidehiko,

Nakayama Masami,

Kuribayashi Futoshi,

ImajohOhmi Shinobu,

Nishitoh Hideki,

Takami Yasunari,

Nakayama Tatsuo

Publication year - 2014

Publication title -

journal of leukocyte biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.819

H-Index - 191

eISSN - 1938-3673

pISSN - 0741-5400

DOI - 10.1189/jlb.0413232

Subject(s) - biology , chromatin , epigenetics , histone , chromatin immunoprecipitation , regulation of gene expression , transcription factor , microbiology and biotechnology , p300 cbp transcription factors , ectopic expression , gene expression , transcriptional regulation , gene , genetics , histone acetyltransferases , promoter

Histone acetyltransferase GCN5 plays key roles in cell differentiation via controlling IRF‐4 gene expression, followed by Blimp‐1 transcriptional activation in B lymphocytes.

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