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Bcl‐2 overexpression ameliorates immune complex‐mediated arthritis by altering FcγRIIb expression and monocyte homeostasis
Author(s) -
Lawlor Kate E.,
Nieuwenhuijze Annemarie,
Parker Karen L.,
Drake Sarah F.,
Campbell Ian K.,
Smith Scott D.,
Vince James E.,
Strasser Andreas,
Wicks Ian P.
Publication year - 2013
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0412190
Subject(s) - arthritis , monocytosis , immunology , immune system , monocyte , biology , autoimmunity , haematopoiesis , autoimmune disease , myeloid , inflammatory arthritis , antibody , cancer research , microbiology and biotechnology , bone marrow , stem cell
Selective effects of the Bcl‐2 survival pathway on monocyte differentiation and its importance in determining the outcome of autoimmune disease.
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