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CCL2/MCP‐1 controls parasite burden, cell infiltration, and mononuclear activation during acute Trypanosoma cruzi infection
Author(s) -
Paiva Cláudia N.,
Figueiredo Rodrigo T.,
KrollPalhares Karina,
Silva Andrea A.,
Silvério Jaline C.,
Gibaldi Daniel,
Pyrrho Alexandre dos S.,
Benjamim Claudia F.,
LannesVieira Joseli,
Bozza Marcelo T.
Publication year - 2009
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0309187
Subject(s) - ccl2 , trypanosoma cruzi , chemokine , biology , myocarditis , immunology , inflammation , chagas disease , infiltration (hvac) , cd8 , amastigote , immune system , parasite hosting , medicine , leishmania , physics , world wide web , computer science , thermodynamics
CCL2 controls heart inflammation and parasite burden in Trypanosoma cruzi infection through cell recruitment and activation.