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C‐reactive protein‐derived peptide 201–206 inhibits neutrophil adhesion to endothelial cells and platelets through CD32
Author(s) -
El Kebir Driss,
Zhang Ying,
Potempa Lawrence A.,
Wu Yi,
Fournier Alain,
Filep János G.
Publication year - 2011
Publication title -
journal of leukocyte biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.819
H-Index - 191
eISSN - 1938-3673
pISSN - 0741-5400
DOI - 10.1189/jlb.0111032
Subject(s) - peptide , proinflammatory cytokine , platelet , inflammation , immunology , platelet activation , biology , cd16 , biochemistry , pharmacology , antigen , cd3 , cd8
Lys201, Gln203, or Trp205 in the C‐terminal hexapeptide of CRP is required for inhibiting neutrophil adherence to endothelial cells and platelets through FcγRII (CD32).