Lack of RAAS inhibition by high-salt intake is associated with arterial stiffness in hypertensive patients | Zendy

C. Kotliar | Zendy; Pablo Kempny | Zendy; Sergio González | Zendy; Carlos Castellaro | Zendy; Pedro Forcada | Zendy; S. Obregon | Zendy; Elena M. V. de Cavanagh | Zendy; J. Chiabaut Svane | Zendy; María Jesús Casarini | Zendy; Mercedes Rojas | Zendy; Felipe Inserra | Zendy

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Lack of RAAS inhibition by high-salt intake is associated with arterial stiffness in hypertensive patients

Author(s) -

C. Kotliar,

Pablo Kempny,

Sergio González,

Carlos Castellaro,

Pedro Forcada,

S. Obregon,

Elena M. V. de Cavanagh,

J. Chiabaut Svane,

María Jesús Casarini,

Mercedes Rojas,

Felipe Inserra

Publication year - 2014

Publication title -

journal of the renin-angiotensin-aldosterone system

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.457

H-Index - 46

eISSN - 1752-8976

pISSN - 1470-3203

DOI - 10.1177/1470320313503692

Subject(s) - pulse wave velocity , arterial stiffness , medicine , blood pressure , plasma renin activity , aldosterone , endocrinology , renin–angiotensin system , urinary system , sodium , cardiology , chemistry , organic chemistry

The relationship between salt intake, blood pressure and RAAS activation is still controversial, being that both high- and low-salt intakes are associated with cardiovascular events in a J-shaped curve pattern. We hypothesized that different patterns of RAAS response to dietary salt intake among hypertensives could be identified, while vascular damage would be related to high-salt intake plus absence of expected RAAS inhibition.

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