Is Angiostatin Involved in Physiological Foveal Avascularity? | Zendy

Michael R. R. Böhm | Zendy; Florian Hodes | Zendy; Katrin Brockhaus | Zendy; Stephanie Hummel | Zendy; Stefan Schlatt | Zendy; Harutyun Melkonyan | Zendy; Solon Thanos | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Is Angiostatin Involved in Physiological Foveal Avascularity?

Author(s) -

Michael R. R. Böhm,

Florian Hodes,

Katrin Brockhaus,

Stephanie Hummel,

Stefan Schlatt,

Harutyun Melkonyan,

Solon Thanos

Publication year - 2016

Publication title -

investigative ophthalmology and visual science

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.935

H-Index - 218

eISSN - 1552-5783

pISSN - 0146-0404

DOI - 10.1167/iovs.16-19286

Subject(s) - angiostatin , retina , biology , retinal , foveal , glial fibrillary acidic protein , fovea centralis , pathology , immunohistochemistry , neuroscience , angiogenesis , medicine , immunology , genetics , biochemistry

The primate central retina is characterized by an avascular fovea and well-defined perifoveal capillary plexus. Neither blood vessels nor their accompanying astrocytes enter the fovea during any stage of retinal development; a balance of angiogenic and angiostatic factors probably maintains foveal avascularity throughout life. The aim of this study was to identify potentially angiorepulsive factors involved in the development of the avascular primate retinal fovea.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research