Central Role of Protein Kinase A in Promoting Trigeminal Nociception in an In Vivo Model of Temporomandibular Disorders | Zendy

Lindsey Kathleen Koop | Zendy; Jordan Hawkins | Zendy; Lauren E. Cornelison | Zendy; Paul L. Durham | Zendy

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Central Role of Protein Kinase A in Promoting Trigeminal Nociception in an In Vivo Model of Temporomandibular Disorders

Author(s) -

Lindsey Kathleen Koop,

Jordan Hawkins,

Lauren E. Cornelison,

Paul L. Durham

Publication year - 2017

Publication title -

journal of oral and facial pain and headache

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.465

H-Index - 42

eISSN - 2333-0384

pISSN - 2333-0376

DOI - 10.11607/ofph.1803

Subject(s) - nociception , trigeminal ganglion , calcitonin gene related peptide , medicine , temporomandibular joint , stimulation , endocrinology , protein kinase a , kinase , mapk/erk pathway , pharmacology , chemistry , neuropeptide , receptor , neuroscience , pathology , biology , biochemistry , sensory system

To investigate cellular changes in the spinal trigeminal nucleus (STN) and trigeminal ganglion (TG) associated with trigeminal nociception mediated by inflammation in the temporomandibular joint (TMJ).

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