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Central Role of Protein Kinase A in Promoting Trigeminal Nociception in an In Vivo Model of Temporomandibular Disorders
Author(s) -
Lindsey Kathleen Koop,
Jordan Hawkins,
Lauren E. Cornelison,
Paul L. Durham
Publication year - 2017
Publication title -
journal of oral and facial pain and headache
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.465
H-Index - 42
eISSN - 2333-0384
pISSN - 2333-0376
DOI - 10.11607/ofph.1803
Subject(s) - nociception , trigeminal ganglion , calcitonin gene related peptide , medicine , temporomandibular joint , stimulation , endocrinology , protein kinase a , kinase , mapk/erk pathway , pharmacology , chemistry , neuropeptide , receptor , neuroscience , pathology , biology , biochemistry , sensory system
To investigate cellular changes in the spinal trigeminal nucleus (STN) and trigeminal ganglion (TG) associated with trigeminal nociception mediated by inflammation in the temporomandibular joint (TMJ).

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