MiR-26a Mediates Ultraviolet B-Induced Apoptosis by Targeting Histone Methyltransferase EZH2 Depending on Myc Expression | Zendy

Ting Zhang | Zendy; Hua Qian | Zendy; Cui Hu | Zendy; Hui Lu | Zendy; Ji-Bing Li | Zendy; Ya-Fen Wu | Zendy; Wěi Li | Zendy

Open Access

MiR-26a Mediates Ultraviolet B-Induced Apoptosis by Targeting Histone Methyltransferase EZH2 Depending on Myc Expression

Author(s) -

Ting Zhang,

Hua Qian,

Cui Hu,

Hui Lu,

Ji-Bing Li,

Ya-Fen Wu,

Wěi Li

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000481759

Subject(s) - hacat , ezh2 , apoptosis , carcinogenesis , cancer research , downregulation and upregulation , histone methyltransferase , uvb induced apoptosis , biology , methyltransferase , microbiology and biotechnology , histone , chemistry , cell culture , cancer , programmed cell death , methylation , gene , genetics , caspase

Ultraviolet B (UVB) damage is the most essential etiological factor in skin carcinogenesis, and apoptosis leads to the efficient elimination of UVB-damaged cells. However, the mechanisms underlying resistance to UVB-induced apoptosis remain unclear.

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