Endogenous Parathyroid Hormone Promotes Fracture Healing by Increasing Expression of BMPR2 through cAMP/PKA/CREB Pathway in Mice | Zendy

Wei Zhou | Zendy; Lipeng Yu | Zendy; Fan Jin | Zendy; Bowen Wan | Zendy; Tao Jiang | Zendy; Jian Yin | Zendy; Yao Huang | Zendy; Qingqing Li | Zendy; Guoyong Yin | Zendy; Zhaoxing Hu | Zendy

Open Access

Endogenous Parathyroid Hormone Promotes Fracture Healing by Increasing Expression of BMPR2 through cAMP/PKA/CREB Pathway in Mice

Author(s) -

Wei Zhou,

Lipeng Yu,

Fan Jin,

Bowen Wan,

Tao Jiang,

Jian Yin,

Yao Huang,

Qingqing Li,

Guoyong Yin,

Zhaoxing Hu

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000477605

Subject(s) - medicine , endocrinology , cyclic adenosine monophosphate , creb , parathyroid hormone , osteoblast , runx2 , chemistry , bone morphogenetic protein 2 , protein kinase a , signal transduction , bone morphogenetic protein , alkaline phosphatase , adenosine , receptor , biology , phosphorylation , transcription factor , calcium , biochemistry , enzyme , gene , in vitro

Endogenous parathyroid hormone (PTH) plays an important role in fracture healing. This study investigated whether endogenous PTH regulates fracture healing by bone morphogenetic protein (BMP) and/or the transforming growth factor-β (TGF-β) signaling pathway.

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