Retrosplenial Cortex (BA 29) Volumes in Behavioral Variant Frontotemporal Dementia and Alzheimer’s Disease | Zendy

Rachel H. Tan | Zendy; Stephanie Wong | Zendy; John R. Hodges | Zendy; Glenda M. Halliday | Zendy; Michael Hornberger | Zendy

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Retrosplenial Cortex (BA 29) Volumes in Behavioral Variant Frontotemporal Dementia and Alzheimer’s Disease

Author(s) -

Rachel H. Tan,

Stephanie Wong,

John R. Hodges,

Glenda M. Halliday,

Michael Hornberger

Publication year - 2013

Publication title -

dementia and geriatric cognitive disorders

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.026

H-Index - 110

eISSN - 1421-9824

pISSN - 1420-8008

DOI - 10.1159/000346392

Subject(s) - chromatin structure remodeling (rsc) complex , retrosplenial cortex , frontotemporal dementia , alzheimer's disease , atrophy , psychology , neuroscience , dementia , hippocampus , cortex (anatomy) , posterior cortical atrophy , cingulate cortex , disease , medicine , pathology , chemistry , central nervous system , biochemistry , nucleosome , gene , histone

The retrosplenial cortex (RSC) is a crucial transit region between the hippocampus and cingulate cortex and has been implicated in spatial navigation and memory. Importantly, RSC atrophy is a predilection site of Alzheimer's (AD) pathology, but there have been no studies assessing structural changes in the RSC in behavioral variant frontotemporal dementia (bvFTD).

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