GH3B6 Pituitary Tumor Cell Proliferation is Mediated by PKCα and PKCε via ERK 1/2-dependent Pathway | Zendy

Juan Pablo Petiti | Zendy; Silvina Gutiérrez | Zendy; Ana Lucía De Paul | Zendy; Verónica Andreoli | Zendy; Claudia Mariela Palmeri | Zendy; Liliana del Valle Sosa | Zendy; José Luís Bocco | Zendy; Alicia Inés Torres | Zendy

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GH3B6 Pituitary Tumor Cell Proliferation is Mediated by PKCα and PKCε via ERK 1/2-dependent Pathway

Author(s) -

Juan Pablo Petiti,

Silvina Gutiérrez,

Ana Lucía De Paul,

Verónica Andreoli,

Claudia Mariela Palmeri,

Liliana del Valle Sosa,

José Luís Bocco,

Alicia Inés Torres

Publication year - 2010

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000320519

Subject(s) - protein kinase c , cell growth , protein kinase b , cell cycle , microbiology and biotechnology , mapk/erk pathway , phorbol , biology , phosphorylation , chemistry , cell , biochemistry

In this report, we explored the role of PKCalpha and PKCe as mediators of phorbol 12-myristate13-acetate (PMA)-induced proliferation in pituitary tumor GH3B6 cells, and determined if the ERK1/2 and Akt pathways were activated.

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