Fractalkine-Induced Endothelial Cell Migration Requires MAP Kinase Signaling | Zendy

Michael V. Volin | Zendy; Nha Huynh | Zendy; Karolina Kłosowska | Zendy; Rosemary D. Reyes | Zendy; James M. Woods | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Fractalkine-Induced Endothelial Cell Migration Requires MAP Kinase Signaling

Author(s) -

Michael V. Volin,

Nha Huynh,

Karolina Kłosowska,

Rosemary D. Reyes,

James M. Woods

Publication year - 2010

Publication title -

pathobiology

Language(s) - Uncategorized

Resource type - Journals

SCImago Journal Rank - 0.941

H-Index - 53

eISSN - 1423-0291

pISSN - 1015-2008

DOI - 10.1159/000272949

Subject(s) - angiogenesis , mapk/erk pathway , microbiology and biotechnology , kinase , phosphorylation , cancer research , biology , signal transduction , cell migration , actin cytoskeleton , cytoskeleton , immunology , cell , biochemistry

Angiogenesis is a well-established characteristic in the rheumatoid arthritis (RA) synovial pannus. We have previously demonstrated that fractalkine (Fkn/ CX3CL1) expression is significantly increased in the RA joint and that fractalkine induces angiogenesis. In this work we studied mechanisms through which Fkn functions as an angiogenic mediator.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research