Open AccessBCR-ABL Truncation due to Premature Translation Termination as a Mechanism of Resistance to Kinase Inhibitors
Author(s) -
Wanlong Ma,
Hagop M. Kantarjian,
Chen-Hsiung Yeh,
Zhong J. Zhang,
Jorge E. Cortés,
Maher Albitar
Publication year - 2009
Publication title -
acta haematologica
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.574
H-Index - 56
eISSN - 1421-9662
pISSN - 0001-5792
DOI - 10.1159/000210060
Subject(s) - nilotinib , dasatinib , imatinib mesylate , philadelphia chromosome , abl , chronic myelogenous leukemia , tyrosine kinase , imatinib , breakpoint cluster region , biology , protein kinase domain , cancer research , chromosomal translocation , microbiology and biotechnology , chemistry , genetics , gene , leukemia , signal transduction , mutant , myeloid leukemia
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