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Nearly Complete Genome Sequence of Brugia malayi Strain FR3
Author(s) -
Alan Tracey,
Jeremy M. Foster,
Michael Paulini,
Alexandra Grote,
John Mattick,
Yu Chih Tsai,
Matthew Chung,
James A. Cotton,
Tyson A. Clark,
Adam Geber,
Nancy Holroyd,
Jonas Korlach,
Silvia Libro,
Sara Lustigman,
Michelle L. Michalski,
Matthew B. Rogers,
Alan Twaddle,
Julie C. Dunning Hotopp,
Matthew Berriman,
Elodie Ghedin
Publication year - 2020
Publication title -
microbiology resource announcements
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.383
H-Index - 35
ISSN - 2576-098X
DOI - 10.1128/mra.00154-20
Subject(s) - brugia malayi , biology , lymphatic filariasis , genome , elephantiasis , whole genome sequencing , chromosome , genetics , autosome , sequence (biology) , filariasis , virology , gene , helminths , zoology
Lymphatic filariasis affects ∼120 million people and can result in elephantiasis and hydrocele. Here, we report the nearly complete genome sequence of the best-studied causative agent of lymphatic filariasis, Brugia malayi The assembly contains four autosomes, an X chromosome, and only eight gaps but lacks a contiguous sequence for the known Y chromosome.

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