z-logo
open-access-imgOpen Access
Podocyte-specific KLF4 is required to maintain parietal epithelial cell quiescence in the kidney
Author(s) -
Jesse Pace,
Robert Bronstein,
Yiqing Guo,
Yaqi Yang,
Chelsea C. Estrada,
Nehaben A. Gujarati,
David J. Salant,
John D. Haley,
Agnieszka B. Bialkowska,
Vincent W. Yang,
John Cijiang He,
Sandeep K. Mallipattu
Publication year - 2021
Publication title -
science advances
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 5.928
H-Index - 146
ISSN - 2375-2548
DOI - 10.1126/sciadv.abg6600
Subject(s) - podocyte , podocalyxin , microbiology and biotechnology , klf4 , kidney glomerulus , kidney , biology , cell , epithelium , neuroscience , pathology , medicine , glomerulonephritis , proteinuria , endocrinology , transcription factor , biochemistry , gene , sox2 , genetics
Integrative omics identifies mediators between podocytes and parietal epithelial cells in proliferative glomerulopathies.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.
Having issues? You can contact us here
Accelerating Research

Address

John Eccles House
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom